genepattern software Search Results


genepattern tools  (Broad Institute Inc)
90
Broad Institute Inc genepattern tools
Genepattern Tools, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/genepattern tools/product/Broad Institute Inc
Average 90 stars, based on 1 article reviews
genepattern tools - by Bioz Stars, 2026-03
90/100 stars
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genepattern 3.5.0 software  (Broad Institute Inc)
90
Broad Institute Inc genepattern 3.5.0 software
Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of <t>GenePattern</t> 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.
Genepattern 3.5.0 Software, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/genepattern 3.5.0 software/product/Broad Institute Inc
Average 90 stars, based on 1 article reviews
genepattern 3.5.0 software - by Bioz Stars, 2026-03
90/100 stars
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collapsedataset module of genepattern software  (Broad Institute Inc)
90
Broad Institute Inc collapsedataset module of genepattern software
Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of <t>GenePattern</t> 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.
Collapsedataset Module Of Genepattern Software, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/collapsedataset module of genepattern software/product/Broad Institute Inc
Average 90 stars, based on 1 article reviews
collapsedataset module of genepattern software - by Bioz Stars, 2026-03
90/100 stars
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genepattern tools  (GraphPad Software Inc)
90
GraphPad Software Inc genepattern tools
Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of <t>GenePattern</t> 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.
Genepattern Tools, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/genepattern tools/product/GraphPad Software Inc
Average 90 stars, based on 1 article reviews
genepattern tools - by Bioz Stars, 2026-03
90/100 stars
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genepattern software package  (Rosetta Biosoftware)
90
Rosetta Biosoftware genepattern software package
Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of <t>GenePattern</t> 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.
Genepattern Software Package, supplied by Rosetta Biosoftware, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/genepattern software package/product/Rosetta Biosoftware
Average 90 stars, based on 1 article reviews
genepattern software package - by Bioz Stars, 2026-03
90/100 stars
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genepattern software package  (Ingenuity Systems)
90
Ingenuity Systems genepattern software package
Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of <t>GenePattern</t> 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.
Genepattern Software Package, supplied by Ingenuity Systems, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/genepattern software package/product/Ingenuity Systems
Average 90 stars, based on 1 article reviews
genepattern software package - by Bioz Stars, 2026-03
90/100 stars
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conversion software genepattern  (Broad Institute Inc)
90
Broad Institute Inc conversion software genepattern
Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of <t>GenePattern</t> 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.
Conversion Software Genepattern, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/conversion software genepattern/product/Broad Institute Inc
Average 90 stars, based on 1 article reviews
conversion software genepattern - by Bioz Stars, 2026-03
90/100 stars
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genepattern software 3.2.1  (Broad Institute Inc)
90
Broad Institute Inc genepattern software 3.2.1
Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of <t>GenePattern</t> 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.
Genepattern Software 3.2.1, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/genepattern software 3.2.1/product/Broad Institute Inc
Average 90 stars, based on 1 article reviews
genepattern software 3.2.1 - by Bioz Stars, 2026-03
90/100 stars
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Image Search Results


Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of GenePattern 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Article Title: CCR5 Knockout Prevents Neuronal Injury and Behavioral Impairment Induced in a Transgenic Mouse Model by a CXCR4-using HIV-1 Glycoprotein 120 1

doi: 10.4049/jimmunol.1302915

Figure Lengend Snippet: Canonical pathways affected by differential expression of the core of common set of genes. CCR5WT HIVgp120tg mice of lines 1 and 2: A, Role of Macrophages, Fibroblasts and Endothelial Cells in Rheumatoid Arthritis (MΦ, Fib, EC in RA); B, Pathogenesis of Multiple Sclerosis (MS); C, Cytotoxic T Lymphocyte-mediated Apoptosis of Target Cells (CTL); D, NFkB Activation by Viruses (NFkB); E, CCR5 Signaling in Macrophages (CCR5 in MΦ). CCR5WT & KO HIVgp120tg mice of lines 1 and 2: F, Communication between Innate and Adaptive Immune Cells (Inn./Adapt. Cells); G, Hepatic Fibrosis / Hepatic Stellate Cell Activation (Hep. Fibrosis); H, Interferon Signaling (IFN); I, Type I Diabetes Mellitus Signaling (T1DM); J, Triggering Receptor Expressed on Monocytes/Myeloid cells 1 (TREM1) Signaling. CCR5KO HIVgp120tg mice of lines 1 and 2: K, GABA Receptor Signaling (GABAR); L, LPS/IL-1 Mediated Inhibition of RXR Function (LPS/IL-1/RXR); M, Mineralocorticoid Biosynthesis (MC); N, Huntington’s Disease Signaling (HD); O, Clathrin-mediated Endocytosis Signaling (Endocytosis). See Table III for P-values, number of molecules affected, molecules in pathway and ratio. The data were filtered on fold-change across the time course by using the online tool and database of IPA for canonical pathways. Based on the filter results, gene lists were generated for mouse lines 1 and 2 and their expression patterns are shown as heatmaps, which were produced using the tool HierarchicalClusteringImage and the default color map of GenePattern 3.5.0 software (Broad Institute) with the data being hierarchically clustered by rows and row normalized. The ‘control’ columns represent the normalized expression levels for the genes in the respective WT/CCR5KO controls.

Article Snippet: Canonical pathway analyses were generated through the use of IPA and heatmaps using the tool HierarchicalClusteringImage with the default color map of GenePattern 3.5.0 software (Broad Institute).

Techniques: Quantitative Proteomics, Activation Assay, Inhibition, Generated, Expressing, Produced, Software, Control